Acne Vulgaris
Acne Vulgaris ( 15 Questions)
A client with Acne Vulgaris is seeking information about the pathophysiology of their condition. Which statement accurately describes the pathophysiology of Acne Vulgaris?
Increased androgen production during puberty does not cause hair follicles to shrink in Acne Vulgaris.
Androgens can stimulate the sebaceous glands to produce more oil, which can contribute to the development of acne, but the shrinking of hair follicles is not a characteristic feature of the condition.
Excessive sebum production is a key factor in the pathophysiology of Acne Vulgaris.
When the sebaceous glands produce an excessive amount of oil (sebum), it can mix with dead skin cells and clog hair follicles, leading to the formation of comedones (blackheads and whiteheads).
Acne Vulgaris is not primarily caused by an overgrowth of yeast on the skin.
While there are various factors involved in acne development, yeast overgrowth is not a primary driver of the condition.
It is more related to sebum production, inflammation, and bacterial colonization.
Hormonal imbalances do play a role in the pathophysiology of Acne Vulgaris, but they do not result in decreased keratinization of hair follicles.
Instead, hormonal imbalances can lead to increased sebum production and alterations in the skin's response to inflammation, contributing to acne development.
Choice A rationale:
Increased androgen production during puberty does not cause hair follicles to shrink in Acne Vulgaris.
Androgens can stimulate the sebaceous glands to produce more oil, which can contribute to the development of acne, but the shrinking of hair follicles is not a characteristic feature of the condition.
Choice B rationale:
Excessive sebum production is a key factor in the pathophysiology of Acne Vulgaris.
When the sebaceous glands produce an excessive amount of oil (sebum), it can mix with dead skin cells and clog hair follicles, leading to the formation of comedones (blackheads and whiteheads).
Choice C rationale:
Acne Vulgaris is not primarily caused by an overgrowth of yeast on the skin.
While there are various factors involved in acne development, yeast overgrowth is not a primary driver of the condition.
It is more related to sebum production, inflammation, and bacterial colonization.
Choice D rationale:
Hormonal imbalances do play a role in the pathophysiology of Acne Vulgaris, but they do not result in decreased keratinization of hair follicles.
Instead, hormonal imbalances can lead to increased sebum production and alterations in the skin's response to inflammation, contributing to acne development.