Obsessive-compulsive disorders

Obsessive-compulsive disorders ( 36 Questions)

A nurse is discussing pharmacological interventions for treating OCDs with a group of patients. Which statement accurately describes the role of selective serotonin reuptake inhibitors (SSRIs) in OCD treatment?


Correct Answer: B

Choice A rationale:

"SSRIs primarily target glutamate modulation in the brain." This statement is inaccurate. Selective serotonin reuptake inhibitors (SSRIs) primarily target the modulation of serotonin levels in the brain, not glutamate. These medications work by inhibiting the reuptake of serotonin, which increases the availability of serotonin in the synaptic cleft and helps regulate mood and anxiety.

Choice B rationale:

"SSRIs work by increasing the availability of serotonin in the brain to reduce obsessions and compulsions." This statement accurately describes the role of SSRIs in treating OCD. Serotonin is a neurotransmitter that plays a role in mood regulation, and imbalances in serotonin have been implicated in OCD. By increasing the availability of serotonin in the brain, SSRIs can help reduce the frequency and intensity of obsessions and compulsions.

Choice C rationale:

"SSRIs are used to enhance positive emotional outcomes in individuals with OCD." This statement is not accurate. While SSRIs can improve mood and reduce anxiety, their primary role in treating OCD is to alleviate the symptoms of obsessions and compulsions by affecting neurotransmitter levels. They are not specifically used to enhance positive emotional outcomes.

Choice D rationale:

"SSRIs are reserved for individuals who have ego-syntonic obsessions and compulsions." This statement is incorrect. Ego-syntonic obsessions and compulsions are those that are consistent with a person's self-image and beliefs, and individuals may not feel a strong need to resist or change them. SSRIs are used to treat both ego-dystonic (inconsistent with self-image) and ego-syntonic obsessions and compulsions in OCD, as these medications target the underlying neurochemical imbalances that contribute to the disorder's symptoms.




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