Paget’s Disease
Paget’s Disease ( 45 Questions)
A client with Paget's disease complains of bone pain that worsens with activity. What is the underlying cause of this pain?
Decreased blood flow to bones: Paget's disease does not typically cause decreased blood flow to bones. Instead, the condition leads to increased vascularity in affected bones.
Bone infarction: Bone infarction (death of bone tissue due to inadequate blood supply) is not a characteristic feature of Paget's disease. Microfractures and inflammation are more common causes of bone pain in this condition.
The bone pain experienced by clients with Paget's disease is often due to microfractures and inflammation in the affected bones. The increased bone turnover and remodeling lead to weaker bones, making them more susceptible to microfractures. The inflammation at the site of these microfractures contributes to the pain, which worsens with activity.
Compression of nerve roots: While nerve compression can cause bone pain, it is not a primary mechanism of pain in Paget's disease. The pain is primarily related to bone remodeling and inflammation.
The bone pain experienced by clients with Paget's disease is often due to microfractures and inflammation in the affected bones. The increased bone turnover and remodeling lead to weaker bones, making them more susceptible to microfractures. The inflammation at the site of these microfractures contributes to the pain, which worsens with activity.
a. Decreased blood flow to bones: Paget's disease does not typically cause decreased blood flow to bones. Instead, the condition leads to increased vascularity in affected bones.
b. Bone infarction: Bone infarction (death of bone tissue due to inadequate blood supply) is not a characteristic feature of Paget's disease. Microfractures and inflammation are more common causes of bone pain in this condition.
d. Compression of nerve roots: While nerve compression can cause bone pain, it is not a primary mechanism of pain in Paget's disease. The pain is primarily related to bone remodeling and inflammation.