Polycystic Ovary Syndrome (PCOS)
Polycystic Ovary Syndrome (PCOS) ( 14 Questions)
What statement by the client indicates a correct understanding of PCOS pathogenesis?
"PCOS is caused by excessive follicle-stimulating hormone (FSH) levels." This statement is not accurate. PCOS is characterized by an imbalance in the ratio of luteinizing hormone (LH) to follicle-stimulating hormone (FSH), with LH often being elevated compared to FSH. Excessive FSH levels are not a primary cause of PCOS.
"Insulin resistance has no impact on the hormonal imbalance in PCOS." This statement is incorrect. Insulin resistance is a significant contributor to the hormonal imbalance seen in PCOS. Insulin resistance can lead to hyperinsulinemia, which in turn can stimulate the ovaries to produce more androgens (male hormones). These elevated androgen levels disrupt normal ovulation and contribute to the formation of ovarian cysts, which are key features of PCOS.
"Elevated androgens disrupt normal ovulation and contribute to cyst formation." This statement is correct. Elevated androgens (such as testosterone) are a hallmark of PCOS and are associated with the disruption of normal ovulation and the development of cysts in the ovaries. These hormonal imbalances are central to the pathogenesis of PCOS.
"PCOS is primarily related to a deficiency of luteinizing hormone (LH)." This statement is not accurate. PCOS is not primarily related to a deficiency of LH but rather to an elevated LH-to-FSH ratio, as mentioned earlier. The hormonal dysregulation in PCOS involves an excess of LH relative to FSH.
Choice A rationale:
"PCOS is caused by excessive follicle-stimulating hormone (FSH) levels." This statement is not accurate.
PCOS is characterized by an imbalance in the ratio of luteinizing hormone (LH) to follicle-stimulating hormone (FSH), with LH often being elevated compared to FSH.
Excessive FSH levels are not a primary cause of PCOS.
Choice B rationale:
"Insulin resistance has no impact on the hormonal imbalance in PCOS." This statement is incorrect.
Insulin resistance is a significant contributor to the hormonal imbalance seen in PCOS.
Insulin resistance can lead to hyperinsulinemia, which in turn can stimulate the ovaries to produce more androgens (male hormones).
These elevated androgen levels disrupt normal ovulation and contribute to the formation of ovarian cysts, which are key features of PCOS.
Choice C rationale:
"Elevated androgens disrupt normal ovulation and contribute to cyst formation." This statement is correct.
Elevated androgens (such as testosterone) are a hallmark of PCOS and are associated with the disruption of normal ovulation and the development of cysts in the ovaries.
These hormonal imbalances are central to the pathogenesis of PCOS.
Choice D rationale:
"PCOS is primarily related to a deficiency of luteinizing hormone (LH)." This statement is not accurate.
PCOS is not primarily related to a deficiency of LH but rather to an elevated LH-to-FSH ratio, as mentioned earlier.
The hormonal dysregulation in PCOS involves an excess of LH relative to FSH.